Recessive Parkinson's disease
Identifieur interne : 000C59 ( Main/Corpus ); précédent : 000C58; suivant : 000C60Recessive Parkinson's disease
Auteurs : Shin-Ichiro Kubo ; Nobutaka Hattori ; Yoshikuni MizunoSource :
- Movement Disorders [ 0885-3185 ] ; 2006-07.
English descriptors
- KwdEn :
Abstract
Parkinson's disease (PD) is a progressive neurodegenerative disease caused by loss of dopaminergic neurons in the substantia nigra pars compacta. Although the etiology of PD remains unclear, it is now clear that genetic factors contribute to the pathogenesis of the disease. Recently, several causative genes have been identified in monogenic forms of PD. Accumulating evidence indicates that their gene products play important roles in mitochondrial function, oxidative stress response, and the ubiquitin–proteasome system, which are also implicated in sporadic PD, suggesting that these gene products share a common pathway to nigral degeneration in both familial and sporadic PD. Here, we review recent advances in knowledge about genes associated with recessive PD, including parkin, PINK1, and DJ‐1. © 2006 Movement Disorder Society
Url:
DOI: 10.1002/mds.20841
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ISTEX:11A1C677F7100DB04F91F2EF9CE3CBB879261529Le document en format XML
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Although the etiology of PD remains unclear, it is now clear that genetic factors contribute to the pathogenesis of the disease. Recently, several causative genes have been identified in monogenic forms of PD. Accumulating evidence indicates that their gene products play important roles in mitochondrial function, oxidative stress response, and the ubiquitin–proteasome system, which are also implicated in sporadic PD, suggesting that these gene products share a common pathway to nigral degeneration in both familial and sporadic PD. Here, we review recent advances in knowledge about genes associated with recessive PD, including parkin, PINK1, and DJ‐1. © 2006 Movement Disorder Society</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Shin‐Ichiro Kubo MD, PhD</name><affiliations><json:string>Department of Neurology, Juntendo University School of Medicine, Tokyo, Japan</json:string></affiliations></json:item><json:item><name>Nobutaka Hattori MD, PhD</name><affiliations><json:string>Department of Neurology, Juntendo University School of Medicine, Tokyo, Japan</json:string><json:string>Research Institute for Diseases of Old Age, Juntendo University School of Medicine, Tokyo, Japan</json:string></affiliations></json:item><json:item><name>Yoshikuni Mizuno MD</name><affiliations><json:string>Department of Neurology, Juntendo University School of Medicine, Tokyo, Japan</json:string><json:string>Research Institute for Diseases of Old Age, Juntendo University School of Medicine, Tokyo, Japan</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>parkin</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>PINK1</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>DJ‐1</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>recessive Parkinson's disease</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>mitochondria</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>oxidative stress</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>ubiquitin–proteasome system</value></json:item></subject><articleId><json:string>MDS20841</json:string></articleId><language><json:string>eng</json:string></language><abstract>Parkinson's disease (PD) is a progressive neurodegenerative disease caused by loss of dopaminergic neurons in the substantia nigra pars compacta. 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